Lab. Seminar
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" Gut Dysbiosis during Influenza Contributes to Pulmonary Pneumococcal Superinfection through Altered Short-Chain Fatty Acid Production " ÀÔ´Ï´Ù.
ÀÌ ³í¹®¿¡¼ ÀÎÇ÷翣ÀÚ °¨¿°¿¡ µû¶ó gut microbiota °¡ º¯ÇÔÀ» È®ÀÎÇÏ¿´°í,
SCFAÁß acetateÀÇ È¿°ú¿¡ ÁýÁßÇÏ¿© ½ÇÇèÀ» ÁøÇàÇÏ¿´½À´Ï´Ù.
Acetate treatment ÇÏ¿´À» ½Ã alveolar macrophageÀÇ killing activity °¡ Áõ°¡Çϸç
SCFA receptor(FFAR2)¸¦ activation ½ÃÄ×À» °æ¿ì bacterial infection controlÀÌ °¡´ÉÇÔÀ» È®ÀÎÇÏ¿´½À´Ï´Ù.
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"Enteric helminth coinfection enhances host susceptibility to neurotropic flaviviruses via a tuft cell-IL-4 receptor signaling axis"¶ó´Â Á¦¸ñÀÇ ³í¹®À¸·Î He..
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